Thursday December 14th 2017

B Cell Receptor Recognition Of Glatiramer Acetate Is Required For Efficacy In Experimental Autoimmune Encephalomyelitis And Results In Differential…

Conclusions: This data, in combination with previous reports showing an alteration in the cytokine profiles of GA specific B cells, suggest that the mechanism of action of GA on B lymphocytes includes the presentation of GA to T lymphocytes, cytokine production, and interactions with other immune cells. Taken together, these data provide a strong foundation for future exploration of optimal Copaxone responders or synergistic combination therapies with an improved risk: benefit ratio compared to currently approved therapeutics for MS.This research was supported by a grant from Teva Pharmaceutical Industries, Ltd.Disclosure: Dr. Jackson has received research support from Teva Neuroscience. Dr. Selva has nothing to disclose. Dr. Niedzielko has nothing to disclose. Dr. Vollmer has received per…

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B Cell Receptor Recognition Of Glatiramer Acetate Is Required For Efficacy In Experimental Autoimmune Encephalomyelitis And Results In Differential…

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